By Steven M Hollenberg
A concise precis of an important scientific and medical themes pertaining to cardiovascular drugs within the fundamental care atmosphere. Writing in an easy-to-read format-background, analysis, and ailment management-and emphasizing useful concepts of overview and remedy, the authors clarify the elemental mechanisms underlying heart problems states and the remedy thoughts that come up from them. subject matters diversity from acute coronary syndromes, arrhythmias, and hyperlipidemia, to high blood pressure, pericardial illnesses, valvular ailment, and reliable angina. Cardiology in family members perform: a realistic consultant bargains a transparent realizing of modern-day regular methods to cardiovascular health problems and a hugely invaluable source while particular questions come up in the course of the care of sufferers. a unique PDA model of Cardiology in relations perform: A kinfolk advisor can also be on hand.
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ACC/AHA 2002 guideline update for the management of patients with chronic stable angina—summary article: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines (Committee on the Management of Patients With Chronic Stable Angina). Circulation 107:149–158, 2003. 31. Gibson RS, Boden WE, Theroux P, et al. Diltiazem and reinfarction in patients with non-Q-wave myocardial infarction. Results of a double-blind, randomized, multicenter trial. N Engl J Med 315:423–429, 1986.
Bates ER, Stomel RJ, Hochman JS, Ohman EM. The use of intraaortic balloon counterpulsation as an adjunct to reperfusion therapy in cardiogenic shock. Int J Cardiol 65 Suppl 1:S37–42, 1998. 7. Becker RC. Hemodynamic, mechanical, and metabolic determinants of thrombolytic efficacy: a theoretic framework for assessing the limitations of Chapter 1 / Acute Coronary Syndromes 8. 9. 10. 11. 12. 13. 14. 15. 16. 17. 18. 19. 20. 21. 22. 31 thrombolysis in patients with cardiogenic shock. Am Heart J 125:919–929, 1993.
Before instituting an antiarrhythmic agent, reversible causes (hyperthyroidism, hypertension, heart failure, cardiac surgery, pulmonary embolism) need to be addressed. Antiarrhythmic agents are divided into classes according to mechanism of action. Class I agents are sodium channel blockers and prolong the QT interval, leading to the potential for proarrhythmia and torsades de pointes. Class Ia agents, which include quinidine, procainamide, and disopyramide, block potassium channels as well. Quinidine can cause sinus and AV nodal blockade, in addition to its proarrhythmic effects.
